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The Kraus 2014 study reported that adipocyte-specific NNMT knockdown in mice increased adipocyte energy expenditure through a mechanism interpreted as elevated methyl-donor availability for polyamine biosynthesis and for histone methylation that modulates adipocyte gene-expression programs [2]

Clinically, this means that correcting a deficiency often relieves unexplained fatigue and brain fog. (NHS sources report that deficiency leads to extreme tiredness and low energy.) Energy misconception: Its important to stress that B injections are not a stimulant